I had identified this person as "Hoffmeister" - the person has now asked me to identify them as "mythdestroyer".
Please see the following information, showing the Spanish Flu of 1918 was bacterial pneumonia. It was Not a Viral disease, which in turn shows Tamiflu and other Viral medicine the WHO is pushing along with governments of the world, would do No Good!
This is most likely what has been happening in the Ukraine and other parts of the world.
WHO is trying to make something VERY Scary out there for us all to get Upset over - Thinking there is no cure! BUT if they were Honest and came out and said this is a Bacterial Pneumonia that needs to be taken care of with antibiotics immediately - there would be no need to Scare the people by releasing "mutated Swine Flu" and Swine Flu Vaccines!
There will be more information coming, I will release it when I can.
The following information is From a Government study about the 1918 Spanish Flu.
I will be looking into how this could be "sprayed" to affect people.
after a lot of research work I finally found what I was looking for:
An official report of an Institute of the US Government which contains much more TRUTH then any declarations from the
WHO. It seem the WHO is rather a marketing company for the Baxter, GlaxoCline Smith and other profitmakers in vaccines then a
Institution for the Health of the people of the world.
Keep your eye on the very important recommendation by NIAID Director Anthony S. Fauci, M.D. – just not anyone!!!!
It is just what the Ukrainian doctor said in his 3. and last report on http://ukraineplague.blogspot.com/2009http://ukraineplague.blogspot.com//11/final-information-and-part-3-from.html
And what has die WHO prepared in his planning: every diseaese comes from A/H1N1-virus and only selling of vaccines!!!
The myth of the Spanish flu I am further researching and give you further detailed information cause it is important to destroy this myth of the beast “Spanish flu”. The WHO will force at least in autumn 2010 compulsary vaccinations the millions of doses must be sold for the profit of their “advisers” in SAGE (Scientific Advisory Council for Emergencies oder SAGE )
A leading adviser is Professor Sir Roy Anderson, a member of the executive council of GlaxoSmithKline, he get about 134 000 € for this job in a year. He also is adviser of the British goverment.
National Institute of Allergy and
Infectious Diseases (NIAID)
Bacterial Pneumonia Caused Most Deaths in 1918 Influenza Pandemic
Implications for Future Pandemic Planning
The majority of deaths during the influenza pandemic of 1918-1919 were not caused by the influenza virus acting alone, report researchers from the National Institute of Allergy and Infectious Diseases (NIAID), part of the National Institutes of Health. Instead, most victims succumbed to bacterial pneumonia following influenza virus infection. The pneumonia was caused when bacteria that normally inhabit the nose and throat invaded the lungs along a pathway created when the virus destroyed the cells that line the bronchial tubes and lungs.
A future influenza pandemic may unfold in a similar manner, say the NIAID authors, whose paper in the Oct. 1 issue of The Journal of Infectious Diseases is now available online. Therefore, the authors conclude, comprehensive pandemic preparations should include not only efforts to produce new or improved influenza vaccines and antiviral drugs but also provisions to stockpile antibiotics and bacterial vaccines as well.
The work presents complementary lines of evidence from the fields of pathology and history of medicine to support this conclusion. "The weight of evidence we examined from both historical and modern analyses of the 1918 influenza pandemic favors a scenario in which viral damage followed by bacterial pneumonia led to the vast majority of deaths," says co-author NIAID Director Anthony S. Fauci, M.D. "In essence, the virus landed the first blow while bacteria delivered the knockout punch."
NIAID co-author and pathologist Jeffery Taubenberger, M.D., Ph.D., examined lung tissue samples from 58 soldiers who died of influenza at various U. S. military bases in 1918 and 1919. The samples, preserved in paraffin blocks, were re-cut and stained to allow microscopic evaluation. Examination revealed a spectrum of tissue damage "ranging from changes characteristic of the primary viral pneumonia and evidence of tissue repair to evidence of severe, acute, secondary bacterial pneumonia," says Dr. Taubenberger. In most cases, he adds, the predominant disease at the time of death appeared to have been bacterial pneumonia. There also was evidence that the virus destroyed the cells lining the bronchial tubes, including cells with protective hair-like projections, or cilia. This loss made other kinds of cells throughout the entire respiratory tract — including cells deep in the lungs — vulnerable to attack by bacteria that migrated down the newly created pathway from the nose and throat.
In a quest to obtain all scientific publications reporting on the pathology and bacteriology of the 1918-1919 influenza pandemic, Dr. Taubenberger and NIAID co-author David Morens, M.D., searched bibliography sources for papers in any language. They also reviewed scientific and medical journals published in English, French and German, and located all papers reporting on autopsies conducted on influenza victims. From a pool of more than 2,000 publications that appeared between 1919 and 1929, the researchers identified 118 key autopsy series reports. In total, the autopsy series they reviewed represented 8,398 individual autopsies conducted in 15 countries.
The published reports "clearly and consistently implicated secondary bacterial pneumonia caused by common upper respiratory flora in most influenza fatalities," says Dr. Morens. Pathologists of the time, he adds, were nearly unanimous in the conviction that deaths were not caused directly by the then-unidentified influenza virus, but rather resulted from severe secondary pneumonia caused by various bacteria. Absent the secondary bacterial infections, many patients might have survived, experts at the time believed. Indeed, the availability of antibiotics during the other influenza pandemics of the 20th century, specifically those of 1957 and 1968, was probably a key factor in the lower number of worldwide deaths during those outbreaks, notes Dr. Morens.
The cause and timing of the next influenza pandemic cannot be predicted with certainty, the authors acknowledge, nor can the virulence of the pandemic influenza virus strain. However, it is possible that — as in 1918 — a similar pattern of viral damage followed by bacterial invasion could unfold, say the authors. Preparations for diagnosing, treating and preventing bacterial pneumonia should be among highest priorities in influenza pandemic planning, they write. "We are encouraged by the fact that pandemic planners are already considering and implementing some of these actions," says Dr. Fauci.
Visit http://www.PandemicFlu.gov for one-stop access to U.S. Government information on avian and pandemic flu.
NIAID conducts and supports research — at NIH, throughout the United States, and worldwide — to study the causes of infectious and immune-mediated diseases, and to develop better means of preventing, diagnosing and treating these illnesses. News releases, fact sheets and other NIAID-related materials are available on the NIAID Web site at http://www.niaid.nih.gov.
The National Institutes of Health (NIH) — The Nation's Medical Research Agency — includes 27 Institutes and Centers and is a component of the U.S. Department of Health and Human Services. It is the primary federal agency for conducting and supporting basic, clinical and translational medical research, and it investigates the causes, treatments, and cures for both common and rare diseases. For more information about NIH and its programs, visit www.nih.gov.
DM Morens et al. Predominant role of bacterial pneumonia as a cause of death in pandemic influenza: Implications for pandemic influenza preparedness. The Journal of Infectious Diseases DOI: 10.1086/591708
I add here two reports of t he investigations of Taubenberger who reproduced the A/H1N1 –Virus of the Spanish flu:
(search “Taubenberger 1918 on http://www.ncbi.nlm.nih.gov/sites/entrez)
The origin and virulence of the 1918 "Spanish" influenza virus.
Department of Molecular Pathology Armed Forces Institute of Pathology Rockville, Maryland, USA.
The "Spanish" influenza pandemic of 1918-19 caused acute illness in 25-30 percent of the world's population and resulted in the death of up to an estimated 40 million people. Using fixed and frozen lung tissue of 1918 influenza victims, the complete genomic sequence of the 1918 influenza virus has been deduced. Sequence and phylogenetic analysis of the completed 1918 influenza virus genes shows them to be the most avian-like among the mammalian-adapted viruses. This finding supports the hypotheses that (1) the pandemic virus contains genes derived from avian-like influenza virus strains and that (2) the 1918 virus is the common ancestor of human and classical swine H1N1 influenza viruses. The relationship of the 1918 virus with avian influenza viruses is further supported by recent work in which the 1918 hemagglutinin (HA) protein crystal structure was resolved. Neither the 1918 hemagglutinin (HA) nor the neuraminidase (NA) genes possess mutations known to increase tissue tropicity that account for the virulence of other influenza virus strains like A/WSN/33 or the highly pathogenic avian influenza H5 or H7 viruses. Using reverse genetics approaches, influenza virus constructs containing the 1918 HA and NA on a modern human influenza virus background were lethal in mice. The complete 1918 virus was even more virulent in mice. The genotypic basis of this virulence has not yet been elucidated. The complete sequence of the non-structural (NS) gene segment of the 1918 virus was deduced and also tested for the hypothesis that enhanced virulence in 1918 could have been due to type I interferon inhibition by the NS1 protein. Results from these experiments suggest that in human cells the 1918 NS1 is a very effective interferon antagonist, but the 1918 NS1 gene does not have the amino acid change that correlates with virulence in the H5N1 virus strains identified in 1997 in Hong Kong. Sequence analysis of the 1918 pandemic influenza virus is allowing us to test hypotheses as to the origin and virulence of this strain. This information should help elucidate how pandemic influenza virus strains emerge and what genetic features contribute to virulence in humans.
PMID: 17526158 [PubMed - indexed for MEDLINE] Arch Virol Suppl. 2005;(19):101-15.
As you see this was at the Department of Molecular Pathology Armed Forces Institute of Pathology Rockville, Maryland, USA in 2005.
The following report is from 2009 and and it seems Taubenberger left the Armed Forces Institute of Pathology Rockville:
Virology. 2009 Oct 25;393(2):338-45. Epub 2009 Sep 5.
An early 'classical' swine H1N1 influenza virus shows similar pathogenicity to the 1918 pandemic virus in ferrets and mice.
Memoli MJ, Tumpey TM, Jagger BW, Dugan VG, Sheng ZM, Qi L, Kash JC, Taubenberger JK.
Viral Pathogenesis and Evolution Section, Laboratory of Infectious Diseases, National Institutes of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892-3203, USA.
The 1918 pandemic influenza virus has demonstrated significant pathogenicity in animal models and is the progenitor of 'classical' swine and modern seasonal human H1N1 lineages. Here we characterize the pathogenicity of an early 'classical' swine H1N1 influenza A virus isolated in 1931 compared to the pathogenicity of the 1918 pandemic virus and a seasonal H1N1 virus in mice and ferrets. A/Swine/Iowa/31 (Sw31) and the 1918 influenza viruses were uniformly lethal in mice at low doses and produced severe lung pathology. In ferrets, Sw31 and 1918 influenza viruses caused severe clinical disease and lung pathology with necrotizing bronchiolitis and alveolitis. The modern H1N1 virus caused little disease in either animal model. These findings revealed that in these models the virulence factors of the 1918 influenza virus are likely preserved in the Sw31 virus and suggest that early swine viruses may be a good surrogate model to study 1918 virulence and pathogenesis.
PMID: 19733889 [PubMed - indexed for MEDLINE]
J Infect Dis. 2008 Oct 1;198(7):962-70.
Predominant role of bacterial pneumonia as a cause of death in pandemic influenza: implications for pandemic influenza preparedness.
Morens DM, Taubenberger JK, Fauci AS.
National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, USA. email@example.com
Future Microbiol. 2009 Apr;4:269-72.
J Infect Dis. 2008 Oct 1;198(7):945-7.
J Infect Dis. 2009 Mar 15;199(6):913; author reply 913-4.
J Infect Dis. 2009 May 1;199(9):1408-9; author reply 1409-10.
BACKGROUND: Despite the availability of published data on 4 pandemics that have occurred over the past 120 years, there is little modern information on the causes of death associated with influenza pandemics.
METHODS: We examined relevant information from the most recent influenza pandemic that occurred during the era prior to the use of antibiotics, the 1918-1919 "Spanish flu" pandemic. We examined lung tissue (Gewebe) sections obtained during 58 autopsies and reviewed pathologic and bacteriologic data from 109 published autopsy series that described 8398 individual autopsy investigations.
RESULTS: The postmortem samples we examined from people who died of influenza during 1918-1919 uniformly exhibited severe changes indicative of bacterial pneumonia. Bacteriologic and histopathologic results from published autopsy series clearly and consistently implicated secondary bacterial pneumonia caused by common upper respiratory-tract bacteria in most influenza fatalities. CONCLUSIONS: The majority of deaths in the 1918-1919 influenza pandemic likely resulted directly from secondary bacterial pneumonia caused by common upper respiratory-tract bacteria. Less substantial data from the subsequent 1957 and 1968 pandemics are consistent with these findings. If severe pandemic influenza is largely a problem of viral-bacterial copathogenesis, pandemic planning needs to go beyond addressing the viral cause alone (e.g., influenza vaccines and antiviral drugs). Prevention, diagnosis, prophylaxis, and treatment of secondary bacterial pneumonia, as well as stockpiling of antibiotics and bacterial vaccines, should also be high priorities for pandemic planning.
PMID: 18710327 [PubMed - indexed for MEDLINE]
"Thank You" Mythdestroyer for providing this information - I look forward to releasing more Information From you!
**Side Note from me (Sherrie)** - I have been working on investigating a certain person/site/company that seems to have an "inside" track on what the lab results are from WHO. I will not name that person/site/company at this time. BUT I will say - going through the history of releases from this site and it being used as the "main Information" of the "mutations of the Swine Flu" and is quoted by MSM all the time as the primary source of information has made me look a little closer. I had become suspicious, due to this person getting what the lab results are - though NO ONE else gets to view or have access to them.
Also, WHY is this company/site/person the ONE AND ONLY site MSM is willing to quote and release their information? Considering MSM ONLY USES PTB APPROVED INFORMATION! Right there - raised my "something is Not quite right antennas". This person/company/site also has connections with vaccine makers - I have wondered: "Are they placed where they are to be part of the Swine Flu game"?
I have found, others have questioned their position in the past. They will say they against WHO, but then they turn around and compliment them. They release all this information, that no one else seems to be able to see or get a hold of.
I will hopefully have enough evidence/information in the near future to release my findings - laying out - WHY I believe the site/company is actually working with WHO and Vaccine makers in Playing a GAME - and they are Playing Their Part VERY WELL!
Now I will also say - I now believe all of this - has been part of the "Hysteria" of a Pandemic/Epidemic spreading around the world. From WHO's releases of "No Way to Stop the Spread" etc. I have to say too, this site worked to their benefit it seems! BUT NO MORE!! This Site is About Getting TRUTH OUT - Not Propaganda!
The FACTS - AS I Now See them:
1. Something WAS SPRAYED IN UKRAINE - and Other Countries possibly (Poland)
2. What was Sprayed - Looks to have been Bacterial in Form -
3. It can Be Taken Care of with Antibiotics - NOT TAMIFLU AND NOT A SWINE FLU VACCINE!
4. A Website - that Releases Information of "mutated Swine Flu" evidence etc. is in with the WHO /Vaccine makers/PTB and Part of the Game.
5. The Swine Flu on a Whole is NOT LETHAL - SO DO NOT TAKE THE VACCINE!
The Vaccine ITSELF IS LETHAL - Look at ALL The Deaths and Adverse Side Effects from it - Though WHO and government agencies Deny the Vaccine has caused them! Look At the Percentage of Deaths and Problems from the Vaccine, compared to the Deaths and Problems from the Swine Flu! You will find the Vaccine has a HIGHER PERCENTAGE than the Swine Flu! It is 1976 All over again!