Swine Flu Accidentally Resurrected From the Dead? 12/10/09 7:33am

Swine Flu Accidentally Resurrected From the Dead?

This was Not an accident and the article hints at that at the end of it.

Article:

Amid an unsettling report today of Tamiflu resistance in a Danish A(H1N1) patient, comes a study in The New England Journal of Medicine tracing the swine flu's 90-year evolution.

The current flu strain has genetic roots in an illness that sickened pigs at a swine show in 1918 in Cedar Rapids, Iowa. A near-century of development since then may include this flu's accidental resurrection from an extinct strain.

Here's what likely went down. At the same time the 1918 flu pandemic was spreading among humans, pigs were hit with a similar respiratory illness. Early experiments confirmed the 1918 swine virus and a human strain emerged about the same time.

According to the authors of the new paper, there was a temporary "extinction" of this strain of virus from humans in 1957. But then it reemerged 20 years later in a small 230-person outbreak in 1976 among soldiers in Fort Dix, New Jersey. That outbreak did not extend beyond the military base.

However the next year H1N1 reemerged in people in the Soviet Union, Hong Kong, and northeastern China. The genetic origin of that 1977 strain turns out not to be the 1976 Fort Dix strain. Instead, it was closely related to a 1950 human strain.

Which means that given the genetic similarity of the two strains, reemergence was likely due to an accidental release during laboratory studies of the 1950 strain that had been preserved as a "freezer" virus.

Ouch. Hate it when that happens.

The authors hypothesize that concerns about the Fort Dix outbreak stimulated a flurry of research on H1N1 viruses in 1976, which led to an accidental release and reemergence of the previously extinct virus a year later. The reemerged 1977 H1N1 strain has been circulating in various seasonal influenzas ever since—including today's.


Or maybe it wasn't such an accidental a release? Conspiracists, restart your engines.

Important Information About 1918 Spanish Flu Found - It was Bacterial Pneumonia 12/04/09 7:10am

Important Information About 1918 Spanish Flu Found - It was Bacterial Pneumonia

I have received this information from the "mythdestroyer" - This person is out of Germany and this is one of a few information pieces I will be posting, sent to me.

I had identified this person as "Hoffmeister" - the person has now asked me to identify them as "mythdestroyer".


Please see the following information, showing the Spanish Flu of 1918 was bacterial pneumonia. It was Not a Viral disease, which in turn shows Tamiflu and other Viral medicine the WHO is pushing along with governments of the world, would do No Good!


This is most likely what has been happening in the Ukraine and other parts of the world.


WHO is trying to make something VERY Scary out there for us all to get Upset over - Thinking there is no cure! BUT if they were Honest and came out and said this is a Bacterial Pneumonia that needs to be taken care of with antibiotics immediately - there would be no need to Scare the people by releasing "mutated Swine Flu" and Swine Flu Vaccines!


There will be more information coming, I will release it when I can.


The following information is From a Government study about the 1918 Spanish Flu.

WE NEED TO NOW - Demand from WHO to be Honest about What is Really Happening - Stop Saying: the Mutated Swine Flu of D225G - Call it by it's name: Bacterial Pneumonia!

I will be looking into how this could be "sprayed" to affect people.

Information:

Dear Sherrie
after a lot of research work I finally found what I was looking for:

An official report of an Institute of the US Government which contains much more TRUTH then any declarations from the
WHO. It seem the WHO is rather a marketing company for the Baxter, GlaxoCline Smith and other profitmakers in vaccines then a
Institution for the Health of the people of the world.

Keep your eye on the very important recommendation by NIAID Director Anthony S. Fauci, M.D. – just not anyone!!!!

It is just what the Ukrainian doctor said in his 3. and last report on http://ukraineplague.blogspot.com/2009http://ukraineplague.blogspot.com//11/final-information-and-part-3-from.html

And what has die WHO prepared in his planning: every diseaese comes from A/H1N1-virus and only selling of vaccines!!!
The myth of the Spanish flu I am further researching and give you further detailed information cause it is important to destroy this myth of the beast “Spanish flu”. The WHO will force at least in autumn 2010 compulsary vaccinations the millions of doses must be sold for the profit of their “advisers” in SAGE (Scientific Advisory Council for Emergencies oder SAGE )

A leading adviser is Professor Sir Roy Anderson, a member of the executive council of GlaxoSmithKline, he get about 134 000 € for this job in a year. He also is adviser of the British goverment.

By mythdestroyer




National Institute of Allergy and
Infectious Diseases (NIAID)

Bacterial Pneumonia Caused Most Deaths in 1918 Influenza Pandemic
Implications for Future Pandemic Planning

The majority of deaths during the influenza pandemic of 1918-1919 were not caused by the influenza virus acting alone, report researchers from the National Institute of Allergy and Infectious Diseases (NIAID), part of the National Institutes of Health. Instead, most victims succumbed to bacterial pneumonia following influenza virus infection. The pneumonia was caused when bacteria that normally inhabit the nose and throat invaded the lungs along a pathway created when the virus destroyed the cells that line the bronchial tubes and lungs.

A future influenza pandemic may unfold in a similar manner, say the NIAID authors, whose paper in the Oct. 1 issue of The Journal of Infectious Diseases is now available online. Therefore, the authors conclude, comprehensive pandemic preparations should include not only efforts to produce new or improved influenza vaccines and antiviral drugs but also provisions to stockpile antibiotics and bacterial vaccines as well.

The work presents complementary lines of evidence from the fields of pathology and history of medicine to support this conclusion. "The weight of evidence we examined from both historical and modern analyses of the 1918 influenza pandemic favors a scenario in which viral damage followed by bacterial pneumonia led to the vast majority of deaths," says co-author NIAID Director Anthony S. Fauci, M.D. "In essence, the virus landed the first blow while bacteria delivered the knockout punch."

NIAID co-author and pathologist Jeffery Taubenberger, M.D., Ph.D., examined lung tissue samples from 58 soldiers who died of influenza at various U. S. military bases in 1918 and 1919. The samples, preserved in paraffin blocks, were re-cut and stained to allow microscopic evaluation. Examination revealed a spectrum of tissue damage "ranging from changes characteristic of the primary viral pneumonia and evidence of tissue repair to evidence of severe, acute, secondary bacterial pneumonia," says Dr. Taubenberger. In most cases, he adds, the predominant disease at the time of death appeared to have been bacterial pneumonia. There also was evidence that the virus destroyed the cells lining the bronchial tubes, including cells with protective hair-like projections, or cilia. This loss made other kinds of cells throughout the entire respiratory tract — including cells deep in the lungs — vulnerable to attack by bacteria that migrated down the newly created pathway from the nose and throat.

In a quest to obtain all scientific publications reporting on the pathology and bacteriology of the 1918-1919 influenza pandemic, Dr. Taubenberger and NIAID co-author David Morens, M.D., searched bibliography sources for papers in any language. They also reviewed scientific and medical journals published in English, French and German, and located all papers reporting on autopsies conducted on influenza victims. From a pool of more than 2,000 publications that appeared between 1919 and 1929, the researchers identified 118 key autopsy series reports. In total, the autopsy series they reviewed represented 8,398 individual autopsies conducted in 15 countries.

The published reports "clearly and consistently implicated secondary bacterial pneumonia caused by common upper respiratory flora in most influenza fatalities," says Dr. Morens. Pathologists of the time, he adds, were nearly unanimous in the conviction that deaths were not caused directly by the then-unidentified influenza virus, but rather resulted from severe secondary pneumonia caused by various bacteria. Absent the secondary bacterial infections, many patients might have survived, experts at the time believed. Indeed, the availability of antibiotics during the other influenza pandemics of the 20th century, specifically those of 1957 and 1968, was probably a key factor in the lower number of worldwide deaths during those outbreaks, notes Dr. Morens.

The cause and timing of the next influenza pandemic cannot be predicted with certainty, the authors acknowledge, nor can the virulence of the pandemic influenza virus strain. However, it is possible that — as in 1918 — a similar pattern of viral damage followed by bacterial invasion could unfold, say the authors. Preparations for diagnosing, treating and preventing bacterial pneumonia should be among highest priorities in influenza pandemic planning, they write. "We are encouraged by the fact that pandemic planners are already considering and implementing some of these actions," says Dr. Fauci.

Visit http://www.PandemicFlu.gov for one-stop access to U.S. Government information on avian and pandemic flu.

NIAID conducts and supports research — at NIH, throughout the United States, and worldwide — to study the causes of infectious and immune-mediated diseases, and to develop better means of preventing, diagnosing and treating these illnesses. News releases, fact sheets and other NIAID-related materials are available on the NIAID Web site at http://www.niaid.nih.gov.

The National Institutes of Health (NIH) — The Nation's Medical Research Agency — includes 27 Institutes and Centers and is a component of the U.S. Department of Health and Human Services. It is the primary federal agency for conducting and supporting basic, clinical and translational medical research, and it investigates the causes, treatments, and cures for both common and rare diseases. For more information about NIH and its programs, visit www.nih.gov.

Reference:
DM Morens et al. Predominant role of bacterial pneumonia as a cause of death in pandemic influenza: Implications for pandemic influenza preparedness. The Journal of Infectious Diseases DOI: 10.1086/591708

http://www.nih.gov/news/health/aug2008/niaid-19.htm





I add here two reports of t he investigations of Taubenberger who reproduced the A/H1N1 –Virus of the Spanish flu:
(search “Taubenberger 1918 on http://www.ncbi.nlm.nih.gov/sites/entrez)

The origin and virulence of the 1918 "Spanish" influenza virus.
Taubenberger JK.
Department of Molecular Pathology Armed Forces Institute of Pathology Rockville, Maryland, USA.


The "Spanish" influenza pandemic of 1918-19 caused acute illness in 25-30 percent of the world's population and resulted in the death of up to an estimated 40 million people. Using fixed and frozen lung tissue of 1918 influenza victims, the complete genomic sequence of the 1918 influenza virus has been deduced. Sequence and phylogenetic analysis of the completed 1918 influenza virus genes shows them to be the most avian-like among the mammalian-adapted viruses. This finding supports the hypotheses that (1) the pandemic virus contains genes derived from avian-like influenza virus strains and that (2) the 1918 virus is the common ancestor of human and classical swine H1N1 influenza viruses. The relationship of the 1918 virus with avian influenza viruses is further supported by recent work in which the 1918 hemagglutinin (HA) protein crystal structure was resolved. Neither the 1918 hemagglutinin (HA) nor the neuraminidase (NA) genes possess mutations known to increase tissue tropicity that account for the virulence of other influenza virus strains like A/WSN/33 or the highly pathogenic avian influenza H5 or H7 viruses. Using reverse genetics approaches, influenza virus constructs containing the 1918 HA and NA on a modern human influenza virus background were lethal in mice. The complete 1918 virus was even more virulent in mice. The genotypic basis of this virulence has not yet been elucidated. The complete sequence of the non-structural (NS) gene segment of the 1918 virus was deduced and also tested for the hypothesis that enhanced virulence in 1918 could have been due to type I interferon inhibition by the NS1 protein. Results from these experiments suggest that in human cells the 1918 NS1 is a very effective interferon antagonist, but the 1918 NS1 gene does not have the amino acid change that correlates with virulence in the H5N1 virus strains identified in 1997 in Hong Kong. Sequence analysis of the 1918 pandemic influenza virus is allowing us to test hypotheses as to the origin and virulence of this strain. This information should help elucidate how pandemic influenza virus strains emerge and what genetic features contribute to virulence in humans.

PMID: 17526158 [PubMed - indexed for MEDLINE] Arch Virol Suppl. 2005;(19):101-15.

As you see this was at the Department of Molecular Pathology Armed Forces Institute of Pathology Rockville, Maryland, USA in 2005.

The following report is from 2009 and and it seems Taubenberger left the Armed Forces Institute of Pathology Rockville:


Virology. 2009 Oct 25;393(2):338-45. Epub 2009 Sep 5.
An early 'classical' swine H1N1 influenza virus shows similar pathogenicity to the 1918 pandemic virus in ferrets and mice.

Memoli MJ, Tumpey TM, Jagger BW, Dugan VG, Sheng ZM, Qi L, Kash JC, Taubenberger JK.
Viral Pathogenesis and Evolution Section, Laboratory of Infectious Diseases, National Institutes of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892-3203, USA.

The 1918 pandemic influenza virus has demonstrated significant pathogenicity in animal models and is the progenitor of 'classical' swine and modern seasonal human H1N1 lineages. Here we characterize the pathogenicity of an early 'classical' swine H1N1 influenza A virus isolated in 1931 compared to the pathogenicity of the 1918 pandemic virus and a seasonal H1N1 virus in mice and ferrets. A/Swine/Iowa/31 (Sw31) and the 1918 influenza viruses were uniformly lethal in mice at low doses and produced severe lung pathology. In ferrets, Sw31 and 1918 influenza viruses caused severe clinical disease and lung pathology with necrotizing bronchiolitis and alveolitis. The modern H1N1 virus caused little disease in either animal model. These findings revealed that in these models the virulence factors of the 1918 influenza virus are likely preserved in the Sw31 virus and suggest that early swine viruses may be a good surrogate model to study 1918 virulence and pathogenesis.
PMID: 19733889 [PubMed - indexed for MEDLINE]
J Infect Dis. 2008 Oct 1;198(7):962-70.

Predominant role of bacterial pneumonia as a cause of death in pandemic influenza: implications for pandemic influenza preparedness.
Morens DM, Taubenberger JK, Fauci AS.

National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, USA. dmorens@niaid.nih.gov
Comment in:

Future Microbiol. 2009 Apr;4:269-72.
J Infect Dis. 2008 Oct 1;198(7):945-7.
J Infect Dis. 2009 Mar 15;199(6):913; author reply 913-4.
J Infect Dis. 2009 May 1;199(9):1408-9; author reply 1409-10.
BACKGROUND: Despite the availability of published data on 4 pandemics that have occurred over the past 120 years, there is little modern information on the causes of death associated with influenza pandemics.

METHODS: We examined relevant information from the most recent influenza pandemic that occurred during the era prior to the use of antibiotics, the 1918-1919 "Spanish flu" pandemic. We examined lung tissue (Gewebe) sections obtained during 58 autopsies and reviewed pathologic and bacteriologic data from 109 published autopsy series that described 8398 individual autopsy investigations.

RESULTS: The postmortem samples we examined from people who died of influenza during 1918-1919 uniformly exhibited severe changes indicative of bacterial pneumonia. Bacteriologic and histopathologic results from published autopsy series clearly and consistently implicated secondary bacterial pneumonia caused by common upper respiratory-tract bacteria in most influenza fatalities. CONCLUSIONS: The majority of deaths in the 1918-1919 influenza pandemic likely resulted directly from secondary bacterial pneumonia caused by common upper respiratory-tract bacteria. Less substantial data from the subsequent 1957 and 1968 pandemics are consistent with these findings. If severe pandemic influenza is largely a problem of viral-bacterial copathogenesis, pandemic planning needs to go beyond addressing the viral cause alone (e.g., influenza vaccines and antiviral drugs). Prevention, diagnosis, prophylaxis, and treatment of secondary bacterial pneumonia, as well as stockpiling of antibiotics and bacterial vaccines, should also be high priorities for pandemic planning.
PMID: 18710327 [PubMed - indexed for MEDLINE]

"Thank You" Mythdestroyer for providing this information - I look forward to releasing more Information From you!

**Side Note from me (Sherrie)** - I have been working on investigating a certain person/site/company that seems to have an "inside" track on what the lab results are from WHO. I will not name that person/site/company at this time. BUT I will say - going through the history of releases from this site and it being used as the "main Information" of the "mutations of the Swine Flu" and is quoted by MSM all the time as the primary source of information has made me look a little closer. I had become suspicious, due to this person getting what the lab results are - though NO ONE else gets to view or have access to them.

Also, WHY is this company/site/person the ONE AND ONLY site MSM is willing to quote and release their information? Considering MSM ONLY USES PTB APPROVED INFORMATION! Right there - raised my "something is Not quite right antennas". This person/company/site also has connections with vaccine makers - I have wondered: "Are they placed where they are to be part of the Swine Flu game"?

I have found, others have questioned their position in the past. They will say they against WHO, but then they turn around and compliment them. They release all this information, that no one else seems to be able to see or get a hold of.

I will hopefully have enough evidence/information in the near future to release my findings - laying out - WHY I believe the site/company is actually working with WHO and Vaccine makers in Playing a GAME - and they are Playing Their Part VERY WELL!

Now I will also say - I now believe all of this - has been part of the "Hysteria" of a Pandemic/Epidemic spreading around the world. From WHO's releases of "No Way to Stop the Spread" etc. I have to say too, this site worked to their benefit it seems! BUT NO MORE!! This Site is About Getting TRUTH OUT - Not Propaganda!

The FACTS - AS I Now See them:

1. Something WAS SPRAYED IN UKRAINE - and Other Countries possibly (Poland)

2. What was Sprayed - Looks to have been Bacterial in Form -

3. It can Be Taken Care of with Antibiotics - NOT TAMIFLU AND NOT A SWINE FLU VACCINE!

4. A Website - that Releases Information of "mutated Swine Flu" evidence etc. is in with the WHO /Vaccine makers/PTB and Part of the Game.

5. The Swine Flu on a Whole is NOT LETHAL - SO DO NOT TAKE THE VACCINE!
The Vaccine ITSELF IS LETHAL - Look at ALL The Deaths and Adverse Side Effects from it - Though WHO and government agencies Deny the Vaccine has caused them! Look At the Percentage of Deaths and Problems from the Vaccine, compared to the Deaths and Problems from the Swine Flu! You will find the Vaccine has a HIGHER PERCENTAGE than the Swine Flu! It is 1976 All over again!

An Email From Germany - And Information About The 1918 Flu and Recreation of it Will Be Coming 12/03/09 7:40pm

An Email From Germany About Tamiflu - And Information About The 1918 Flu and Recreation of it Will Be Coming

The Person from Germany, does not want their name used, but I can use the name Hoffmeister for them.

There were two information pieces sent to me, but the person has asked that I wait to post those, until someone else is heard from on them.

He did send his thoughts from his research on Tamiflu and letting me post it - here that is. I will post the other things (which are quite long) when I have been given the "go ahead".

Email:

Somewhere I read in an offical WHO documents that they belief
that the Tamiflu is quickly dropped out of humans and reach the
groundwater and from there it returns to our drinking water.
And the little beasts of swineflu are just waiting in water and there they
become resistent. It is quite curious, but the WHO produces a lot of
nonsence.They are rather a marketing company then a health company!!

Well I write to you because it is very important that Tamiflu cannot work
on bacterial pneumonia of course you have to take antibiotics. Tamiflu resistence is not a question of the swine flu but of an another disease, mostly bacterial pneumonia!

That is what the Ukrainian doctors told us. Taking Tamiflu and waiting might kill
you.

Taking Tamiflu will undermine your immunsystem further.
By the way I just heard from a man in Germany who took the vaccine Pandemrix and after a few weeks later he gots the Swineflu.
I have send the two documents to (I took out the name) for an answer for the
problem Virus-immuncells reaction.

The WHO tells us there is a beast that will kill us, just as they learned
by Charles Darwin - live is a fight - and the strongest survive.
Bruce Lipton tells another story, live is not a fight it is a community of intelligent cells and every cell strives for a comfort enviroment (it is the host= us) and they are distroying themselves if they gone kill the host. Therefore their mutation are not becoming more deadly. The deadly strains are produced in laboratories as I wrote you, the scientists cannot really controll. They are playing with fire and telling us they are doing right for humn health. They don't know it better!!!

All the deaths reported in Germany are caused by severe underlaying diseases
and all the deaths after vaccination are also caused by severe underlaying
diseases. Of course the WHO tells another story: The first are killed by the H1N1-Virus, and the second by underlaying diseases. Well it sounds rather schizophrenic and of course it is. And then the WHO tells the people who have these severe diseases should vaccunated first. And they believe what they are telling mot using their common mind!

Well my conclusion is:
don't be fixed on the beast it is rather a puppy but be aware of other
severe underlaying diseases when you go to the doctors.
In case of bacterial pneumonia no tamiflu but antibiotics.
The description giving by the Ukrainian doctor including the detailled progression
of the disease of "Ukrainian flu" enables the people to react at the right time and call thei doctor. I recommend put this discription every day on the blog!! It might live saving for a lot of people!!
I hope this may helpful for the people and think of St. Louis people 1918-20!!!
Be prepared!!!

**Note** In reference to St. Louis, in a document sent to me - there is a Chart - with St. Louis in it. It shows the 1918 flu DID NOT Kill the people there as it did other places! Why? It seems they Took VITAMINS and protected themselves NATURALLY!! I will be posting it all, at a later date - when Hoffmeister tells me, I can.

All That I am Reading - 1918 Spanish Flu Has Been Recreated and is around the World in Cases 11/23/09 8:45am

All That I am Reading - 1918 Spanish Flu Has Been Recreated and is around the World in Cases

It seems to me the 1918 Spanish Flu was successfully recreated and is around the world showing up in cases as a "mutated Swine Flu". This is what I can gather from many news reports. But, this is not being said as What is going on in the Ukraine.

The CDC, WHO and governments are saying a mutated swine flu resistant to Tamiflu results have been detected in Norway, North Carolina, U.S., Brazil, Japan, Mexico, Ukraine and the U.K.

There are many articles about this today. I am going to insert the links to them.

Here is what Wikipedia says about the 1918 Spanish flu:

Scientists have used tissue samples from frozen victims to reproduce the virus for study. Given the strain's extreme virulence there has been controversy regarding the wisdom of such research. Among the conclusions of this research is that the virus kills via a cytokine storm (overreaction of the body's immune system) which explains its unusually severe nature and the concentrated age profile of its victims. The strong immune systems of young adults ravaged the body, whereas the weaker immune systems of children and middle-aged adults caused fewer deaths.[9]

The second wave of the 1918 pandemic was much deadlier than the first. The first wave had resembled typical flu epidemics; those most at risk were the sick and elderly, while younger, healthier people recovered easily. But in August, when the second wave began in France, Sierra Leone and the United States,[41] the virus had mutated to a much deadlier form. This has been attributed to the circumstances of the First World War.[42] In civilian life evolutionary pressures favour a mild strain: those who get really sick stay home, and those mildly ill continue with their lives, go to work and go shopping, preferentially spreading the mild strain. In the trenches the evolutionary pressures were reversed: soldiers with a mild strain remained where they were, while the severely ill were sent on crowded trains to crowded field hospitals, spreading the deadlier virus. So the second wave began and flu quickly spread around the world again.[43] It was the same flu, in that most of those who recovered from first-wave infections were immune, but it was now far more deadly, and the most vulnerable people were those who were like the soldiers in the trenches—young, otherwise healthy adults.[44] Consequently, during modern pandemics, health officials pay attention when the virus reaches places with social upheaval, looking for deadlier strains of the virus.[43]

One theory is that the virus strain originated at Fort Riley, Kansas, by two genetic mechanisms – genetic drift and antigenic shift – in viruses in poultry and swine which the fort bred for food; the soldiers were then sent from Fort Riley to different places around the world, where they spread the disease. However, evidence from a recent reconstruction of the virus suggests that it jumped directly from birds to humans, without traveling through swine.[55][56] This suggestion is slightly controversial,[57] and other research suggests that the strain originated in a mammalian species.[58]

An effort to recreate the 1918 flu strain (a subtype of avian strain H1N1) was a collaboration among the Armed Forces Institute of Pathology, Southeast Poultry Research Laboratory and Mount Sinai School of Medicine in New York City; the effort resulted in the announcement (on October 5, 2005) that the group had successfully determined the virus's genetic sequence, using historic tissue samples recovered by pathologist Johan Hultin from a female flu victim buried in the Alaskan permafrost and samples preserved from American soldiers.[59]

On January 18, 2007, Kobasa et al. reported that monkeys (Macaca fascicularis) infected with the recreated strain exhibited classic symptoms of the 1918 pandemic and died from a cytokine storm[60]—an overreaction of the immune system. This may explain why the 1918 flu had its surprising effect on younger, healthier people, as a person with a stronger immune system would potentially have a stronger overreaction.[61]

On September 16, 2008, the body of Yorkshireman Sir Mark Sykes was exhumed to study the RNA of the Spanish flu virus in efforts to understand the genetic structure of modern H5N1 bird flu. Sykes had been buried in 1919 in a lead coffin which scientists hope will have helped preserve the virus.[62]

In December 2008, research by Yoshihiro Kawaoka of the University of Wisconsin linked the presence of three specific genes (termed PA, PB1, and PB2) and a nucleoprotein derived from 1918 flu samples to the ability of the flu virus to invade the lungs and cause pneumonia. The combination triggered similar symptoms in animal testing.[63]

The original date of appearance of this virus is not clear. An estimated date for its appearance in mammalian hosts has been put at the period 1882–1913.[64] This ancestor virus diverged about 1913–1915 into two clades which gave rise to the classical swine and human H1N1 influenza lineages. The last common ancestor of human strains dates to between February 1917 and April 1918. Because pigs are more readily infected with avian influenza viruses than are humans, it is likely that they were the original recipient of the virus. This in turn suggests that the virus was introduced into humans sometime between 1913 and 1918.



A 2005 National Geographic Article about How the 1918 Virus was dug up and the U.S. government was reproducing it to "study it".

The purpose was to get at questions relating to the 1918 pandemic," said Jeffery Taubenberger, of the Armed Forces Institute of Pathology (AFIP) in Rockville, Maryland. Taubenberger co-authored one of several related papers in this week's issues of the journals Nature and Science.

"How did this particular virus form and get into humans? How did a pandemic start?" Taubenberger said. "Why was this particular virus so virulent? And in a broader sense what can we learn from the lessons of 1918 that can help us in the future?"

Influenza viruses were unknown in 1918, so there was no way for doctors or scientists to directly study the flu during or after the outbreak.

But some institutions, like the AFIP, preserved tissue samples from 1918 flu victims. Those 87-year-old samples—and others from a victim who was buried in, and preserved by, Alaskan permafrost—yielded tiny fragments of genetic material that were used to piece together the virus's genetic coding signature.

The final genes of the virus's genome sequence are being published this week. Scientists used the completed, full viral sequence to create a live virus with the eight viral genes of the Spanish flu, named for an early, devastating outbreak in Spain.

Even if somehow released, that virus would be unlikely to cause a pandemic like that of 1918, because humans have likely acquired some immunity in the intervening decades. Nonetheless, it is currently contained at Atlanta, Georgia's Centers for Disease Control and Prevention (CDC) under extremely strict security.


I want to make sure People also KNOW - A GROUP OF PEOPLE WHO NEVER GOT THE SPANISH FLU DURING THAT TIME - WERE PEOPLE WHO WORKED IN CINNAMON FACTORIES!!

Please put Cinnamon on your List of "Must Get Items" - along with Vitamins C, D, Garlic and Kombucha Tea.

Some news out today about this "Mutated Swine Flu"

Swine flu virus mutation detected

The Department of Health has found the same mutation in a human swine influenza virus sample as the one detected in Norway recently.

The department has examined the genetic sequence of human swine flu viruses in its monitoring system. Out of the 123 sequences studied, one sample showed the same mutation as the Norway strain.

The virus was taken from a year-old boy who developed flu-like symptoms July 22. He was admitted to Prince of Wales Hospital July 25 and discharged three days later. He has recovered.

Mutations are frequently encountered in influenza viruses. According to the World Health Organisation the same mutation of the virus has been found on the Mainland and in other countries, including Brazil, Japan, Mexico, Ukraine and the US.

The virus with this mutation remained sensitive to antiviral drugs, Tamiflu and Relenza. No evidence suggests these mutations are leading to an unusual increase in the number of human swine flu infections or a greater number of severe or fatal cases.

Tamiflu-resistant H1N1 flu cluster reported in NC

Portion of Article:

ATLANTA — Four North Carolina patients at a single hospital tested positive for a type of swine flu that is resistant to Tamiflu, health officials said Friday.

The cases reported at Duke University Medical Center over six weeks make up the biggest cluster seen so far in the U.S.

Tamiflu -- made by Switzerland's Roche Group -- is one of two flu medicines that help against swine flu, and health officials have been closely watching for signs that the virus is mutating, making the drugs ineffective.

More than 50 resistant cases have been reported in the world since April, including 21 in the U.S. Almost all in the U.S. were isolated, said officials with the U.S. Centers for Disease Control and Prevention.

The BBC reported another cluster of five Tamiflu-resistant cases this week in Wales, in the United Kingdom.

The CDC has sent three disease investigators to North Carolina to help in the investigation there, said Dave Daigle, a CDC spokesman. CDC testing confirmed the Tamiflu-resistant cases.

All links are embedded in their headlines.

As I see it, we have various viruses around the world being spread now. Amazing how this has come at the same time as a Global Meltdown of the Economy!

Read/Research about all the various missing lab samples from military installations, about all the microbiologist that have died over the last few years about all the discrepancies in Big Pharma vaccine information.

After doing research of how many viruses have been detected in the Swine Flu in the beginning - and now the Mutated Virus make your own mind up if all of this "Just happens to have shown up on it's own and if the Virus happened to Mutate on it's own"!

Oh, when will the WHO, CDC and Governments come out and say the "VACCINE IS WORTHLESS NOW" Since the flu has mutated and the Vaccine was NOT created for this "Mutated Strain"?